So glad Ian sent in his article. I had not seen this thread.
I would like to hear what folks have to say about what I was taught by Sahrmann about posture before I became, although somewhat stubbornly, a neuronut.
She doesn't necessarily care if a curve is "abnormal" by 20 degrees or whatever unless she can somehow correlate it with painful positions/movements. For example, if someone has neck or shoulder pain, she will lift the scapula and ask for a difference. If this reduces the pain, the static posture will be significant in determining her "movement impairment syndrome" but if not, she moves on. She determines if this is part of a significant pattern through observing various movements and looking for other correlations.
Now, her explanation for how this has become a problem is what I think we can maybe discuss. I have never heard her make mention of motor control in its relation to lower brain centers, sympathetic/parasympathetic response, and she basically only acknowledges that the problem could be due to "nerve tension." This is obviously a big weakness in the theory of her approach. However, a strength could be in her explanations in regards to function. For example, if a person spends their whole day leaning to the right because the file drawer in the desk is on that side, tissues adapt over time, shortening, possibly resulting in a posture that leans to the right.
If this is the case, the shortening is not protective, but adaptive. Now, the right sided nerve roots are being compressed without relief, and an abnormal neurodynamic may be the result. I believe that Shacklock would classify this as an excessive closing dysfunction on the right.
So, is this abnormal neurodynamic the result of (cause and effect type) an adaptive posture instead of a correlation? Does this adaptation model, based on repetitive, compulsive function, hold up?
Cory
I would like to hear what folks have to say about what I was taught by Sahrmann about posture before I became, although somewhat stubbornly, a neuronut.
She doesn't necessarily care if a curve is "abnormal" by 20 degrees or whatever unless she can somehow correlate it with painful positions/movements. For example, if someone has neck or shoulder pain, she will lift the scapula and ask for a difference. If this reduces the pain, the static posture will be significant in determining her "movement impairment syndrome" but if not, she moves on. She determines if this is part of a significant pattern through observing various movements and looking for other correlations.
Now, her explanation for how this has become a problem is what I think we can maybe discuss. I have never heard her make mention of motor control in its relation to lower brain centers, sympathetic/parasympathetic response, and she basically only acknowledges that the problem could be due to "nerve tension." This is obviously a big weakness in the theory of her approach. However, a strength could be in her explanations in regards to function. For example, if a person spends their whole day leaning to the right because the file drawer in the desk is on that side, tissues adapt over time, shortening, possibly resulting in a posture that leans to the right.
If this is the case, the shortening is not protective, but adaptive. Now, the right sided nerve roots are being compressed without relief, and an abnormal neurodynamic may be the result. I believe that Shacklock would classify this as an excessive closing dysfunction on the right.
So, is this abnormal neurodynamic the result of (cause and effect type) an adaptive posture instead of a correlation? Does this adaptation model, based on repetitive, compulsive function, hold up?
Cory
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