Dysfunctional motor outputs

[Raulan2]

"Abnormal neurodynamics from lack of moving, in iliohypogastric/lateral cutaneous, ilioinguinal, obturator, and genitofemoral nerves, can create such a motor output problem, pelvic obliquity, lack of hip extension etc. Just thought I'd throw in more peripheralist material for fun (ectodermal however). In this case, peripheral drivers to central output, whether pain output or apparent motor dysfunction or both combined."
Diane,
you posted this on the re site and you peaked my interest. If you see pelvic obliquity or limited hip extension (for example) based on lack of moving, please expound on what mechanism you mean by motor output problem? could the lack of moving, also be a neurologically driven mechanism?

[Diane]

could the lack of moving, also be a neurologically driven mechanism? Usually, that's ALL it is..

[Raulan2]

Diane
in your understanding, could that motor output develop patterns? For example, if some event contributed to abnormal neurodynamics that in turn caused hypertonicity of some muscle groups (for a specific habitual movement), and inhibition of the opposing muscle groups ( to allow for a compensated or habitual movent to occur), could that lead to tunnel syndromes?

[Diane]

I expect so. And I expect that one part of the brain doesn't realize it's doing this to another part of the brain. A situation might be, a statically held position starts up a hypoxic signal somewhere, which reaches consciousness, but the person "downregulates" the urge to move (a bad choice). Some part of the brain moves to defend itself/the nerve that is signalling hypoxia.. tension starts up, nerve gets blood flow again, quiets down. But the non-conscious tightening, once useful and relieving, becomes habitual, and some other nerve in the middle of the mesoderm eventually gets affected .. and starts to signal.. That which was once a nonconscious solution through static contraction has become another problem instead.